READING-DISABILITY AND HYPERACTIVITY DISORDER - EVIDENCE FOR A COMMON GENETIC ETIOLOGY

被引:59
作者
LIGHT, JG
PENNINGTON, BF
GILGER, JW
DEFRIES, JC
机构
[1] UNIV MICHIGAN,INST SOCIAL RES,ANN ARBOR,MI 48109
[2] UNIV DENVER,DEPT PSYCHOL,DENVER,CO 80208
[3] UNIV KANSAS,DEPT SPEECH LANGUAGE HEARING SCI & DISORDERS,LAWRENCE,KS 66045
[4] UNIV COLORADO,INST BEHAV GENET,BOULDER,CO 80309
关键词
D O I
10.1080/87565649509540623
中图分类号
B844 [发展心理学(人类心理学)];
学科分类号
040202 ;
摘要
In order to assess the genetic etiology of comorbid reading disability and attention deficit hyperactivity disorder (ADHD), reading performance and hyperactivity data (Diagnostic Interview for Children and Adolescents [DICA]; Herjanic, Campbell, & Reich, 1982) from 61 identical and 43 same-sex fraternal twin pairs tested in the Colorado Reading Project were subjected to multiple-regression analysis (DeFries & Fulker, 1985, 1988). Results suggest that approximately 45% of the proband deficit in reading is due to genetic factors that also influence hyperactivity. Moreover, heritable variation accounted for 70% of the observed covariance between the reading acid hyperactivity measures. Thus, comorbid reading disability and ADHD (as measured by parental ratings on the DICA) is due, at least in part, to heritable influences.
引用
收藏
页码:323 / 335
页数:13
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