THE HEMATOPOIETIC GROWTH-FACTORS INTERLEUKIN-3 AND COLONY STIMULATING FACTOR-I STIMULATE PROLIFERATION BUT DO NOT INDUCE INOSITOL LIPID BREAKDOWN IN MURINE BONE-MARROW-DERIVED MACROPHAGES

被引:86
作者
WHETTON, AD [1 ]
MONK, PN [1 ]
CONSALVEY, SD [1 ]
DOWNES, CP [1 ]
机构
[1] SK&F RES LTD,WELWYN AL6 9AR,HERTS,ENGLAND
关键词
D O I
10.1002/j.1460-2075.1986.tb04640.x
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
引用
收藏
页码:3281 / 3286
页数:6
相关论文
共 38 条
[21]   CALCIUM REGULATION OF PHOSPHATIDYL INOSITOL TURNOVER IN MACROPHAGE ACTIVATION BY FORMYL PEPTIDES [J].
HOLIAN, A ;
STICKLE, DF .
JOURNAL OF CELLULAR PHYSIOLOGY, 1985, 123 (01) :39-45
[22]   TRANSMEMBRANE SIGNALING BY THE T-CELL ANTIGEN RECEPTOR - PERTURBATION OF THE T3-ANTIGEN RECEPTOR COMPLEX GENERATES INOSITOL PHOSPHATES AND RELEASES CALCIUM-IONS FROM INTRACELLULAR STORES [J].
IMBODEN, JB ;
STOBO, JD .
JOURNAL OF EXPERIMENTAL MEDICINE, 1985, 161 (03) :446-456
[23]  
JACKOWSKI S, 1986, J BIOL CHEM, V261, P4978
[24]   STIMULATION OF HEMATOPOIESIS INVIVO BY RECOMBINANT BACTERIAL MURINE INTERLEUKIN-3 [J].
KINDLER, V ;
THORENS, B ;
DEKOSSODO, S ;
ALLET, B ;
ELIASON, JF ;
THATCHER, D ;
FARBER, N ;
VASSALLI, P .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1986, 83 (04) :1001-1005
[25]   EGF AND INSULIN ACTION IN FIBROBLASTS - EVIDENCE THAT PHOSPHOINOSITIDE HYDROLYSIS IS NOT AN ESSENTIAL MITOGENIC SIGNALING PATHWAY [J].
LALLEMAIN, G ;
POUYSSEGUR, J .
FEBS LETTERS, 1986, 197 (1-2) :344-348
[26]  
LORD BI, 1986, LYMPHOKINE RES, V5, P97
[27]  
MACARA IG, 1984, AM J PHYSL, V284, pC3
[28]   THE ROLE OF PROTEIN KINASE-C IN CELL-SURFACE SIGNAL TRANSDUCTION AND TUMOR PROMOTION [J].
NISHIZUKA, Y .
NATURE, 1984, 308 (5961) :693-698
[29]   A HIGH-PERFORMANCE LIQUID-CHROMATOGRAPHIC METHOD TO MEASURE P-32 INCORPORATION INTO PHOSPHORYLATED METABOLITES IN CULTURED-CELLS [J].
SHARPS, ES ;
MCCARL, RL .
ANALYTICAL BIOCHEMISTRY, 1982, 124 (02) :421-424
[30]   THE C-FMS PROTO-ONCOGENE PRODUCT IS RELATED TO THE RECEPTOR FOR THE MONONUCLEAR PHAGOCYTE GROWTH-FACTOR, CSF-1 [J].
SHERR, CJ ;
RETTENMIER, CW ;
SACCA, R ;
ROUSSEL, MF ;
LOOK, AT ;
STANLEY, ER .
CELL, 1985, 41 (03) :665-676