APPARENT MINERALOCORTICOID EXCESS SYNDROMES

[91] Souness G.W., Morris D.J., The antinatriuretic and kaliuretic effects of the glucocorticoids corticosterone and cortisol following pretreatment with carbenoxolone sodium (a liquorice derivative) in the adrenalectomized rat, Endocrinology, 124, (1989)

[92] Stewart P.M., Wallace A.M., Valentino R., Burt D., Shackleton C.H.L., Edwards C.R.W., Mineralocorticoid activity of liquolice: 11-ß-hydroxysteroid dehydrogenase deficiency comes of age, Lancet, 2, (1987)

[93] MacKenzie M.A., Hoefnagels W.H.L., Jansen R.W.M.M., Benraad T.J., Kloppenborg P.W.C., The influence of glycyrrhetinic acid on plasma cortisol and cortisone in healthy young volunteers, J. Clin. Endocrinol. Metab., 70, (1990)

[94] Farese R.V., Biglieri E.G., Shackleton C.H.L., Irony I., Gomez-Fontes R., Licorice-induced hypermineralocorticoidism, N. Engl. J. Med., 325, (1991)

[95] Kageyama Y., Suzuki H., Saruta T., Glycyrrhizin induces mineralocorticoid activity through alterations in cortisol metabolism in the human kidney, J. Endocrinol., 135, (1992)

[96] Souness G.W., Morris D., The “mineralocorticoid-like” actions conferred on corticosterone by carbenoxolone are inhibited by the mineralocorticoid receptor (type I) antagonist RU28318, Endocrinology, 129, (1991)

[97] Whorwood C.B., Sheppard M.C., Stewart P.M., Licorice inhibits 11ß-hydroxysteroid dehydrogenase messenger ribonucleic acid levels and potentiates glucocorticoid hormone action, Endocrinology, 132, (1993)

[98] Stewart P.M., Whorwood C.B., 11ß-hydroxysteroid dehydrogenase activity and corticosteroid hormone action, Steroids, 59, (1994)

[99] Monder C., Corticosteroids, kidneys, sweet roots and dirty drugs, Mol. Cell. Endocrinol., 78, (1991)

[100] Sheppard K., Funder J.W., Mineralocorticoid specificity of renal type I receptors: in vivo binding studies, Am. J. Physiol., 252, (1987)

[91] Souness G.W., Morris D.J., The antinatriuretic and kaliuretic effects of the glucocorticoids corticosterone and cortisol following pretreatment with carbenoxolone sodium (a liquorice derivative) in the adrenalectomized rat, Endocrinology, 124, (1989)

[92] Stewart P.M., Wallace A.M., Valentino R., Burt D., Shackleton C.H.L., Edwards C.R.W., Mineralocorticoid activity of liquolice: 11-ß-hydroxysteroid dehydrogenase deficiency comes of age, Lancet, 2, (1987)

[93] MacKenzie M.A., Hoefnagels W.H.L., Jansen R.W.M.M., Benraad T.J., Kloppenborg P.W.C., The influence of glycyrrhetinic acid on plasma cortisol and cortisone in healthy young volunteers, J. Clin. Endocrinol. Metab., 70, (1990)

[94] Farese R.V., Biglieri E.G., Shackleton C.H.L., Irony I., Gomez-Fontes R., Licorice-induced hypermineralocorticoidism, N. Engl. J. Med., 325, (1991)

[95] Kageyama Y., Suzuki H., Saruta T., Glycyrrhizin induces mineralocorticoid activity through alterations in cortisol metabolism in the human kidney, J. Endocrinol., 135, (1992)

[96] Souness G.W., Morris D., The “mineralocorticoid-like” actions conferred on corticosterone by carbenoxolone are inhibited by the mineralocorticoid receptor (type I) antagonist RU28318, Endocrinology, 129, (1991)

[97] Whorwood C.B., Sheppard M.C., Stewart P.M., Licorice inhibits 11ß-hydroxysteroid dehydrogenase messenger ribonucleic acid levels and potentiates glucocorticoid hormone action, Endocrinology, 132, (1993)

[98] Stewart P.M., Whorwood C.B., 11ß-hydroxysteroid dehydrogenase activity and corticosteroid hormone action, Steroids, 59, (1994)

[99] Monder C., Corticosteroids, kidneys, sweet roots and dirty drugs, Mol. Cell. Endocrinol., 78, (1991)

[100] Sheppard K., Funder J.W., Mineralocorticoid specificity of renal type I receptors: in vivo binding studies, Am. J. Physiol., 252, (1987)