IVGG THERAPY IN KAWASAKI DISEASE - MECHANISM(S) OF ACTION

被引：28

作者：

SHULMAN, ST

机构：

来源：

CLINICAL IMMUNOLOGY AND IMMUNOPATHOLOGY | 1989年 / 53卷 / 02期

关键词：

D O I：

10.1016/0090-1229(89)90079-2

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

引用

页码：S141 / S146

页数：6

共 23 条

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[13] IMMUNOGLOBULIN-M ANTIBODIES PRESENT IN THE ACUTE PHASE OF KAWASAKI SYNDROME LYSE CULTURED VASCULAR ENDOTHELIAL-CELLS STIMULATED BY GAMMA INTERFERON [J].

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[14] 2 MONOKINES, INTERLEUKIN-1 AND TUMOR-NECROSIS-FACTOR, RENDER CULTURED VASCULAR ENDOTHELIAL-CELLS SUSCEPTIBLE TO LYSIS BY ANTIBODIES CIRCULATING DURING KAWASAKI SYNDROME [J].

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[16] ABSENCE OF SIGNIFICANT RNA-DEPENDENT DNA-POLYMERASE ACTIVITY IN LYMPHOCYTES FROM PATIENTS WITH KAWASAKI SYNDROME [J].

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[17] THE TREATMENT OF KAWASAKI SYNDROME WITH INTRAVENOUS GAMMA-GLOBULIN [J].

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[18] ENHANCED NEUTROPHILIC FUNCTIONS IN MUCOCUTANEOUS LYMPH-NODE SYNDROME, WITH SPECIAL REFERENCE TO THE POSSIBLE ROLE OF INCREASED OXYGEN INTERMEDIATE GENERATION IN THE PATHOGENESIS OF CORONARY THROMBOARTERITIS [J].

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[19] IMPAIRED GRANULOCYTE CHEMOTAXIS AND INCREASED CIRCULATING IMMUNE-COMPLEXES IN KAWASAKI DISEASE [J].

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[20] WHAT IS THE STATUS OF INTRAVENOUS GAMMA-GLOBULIN FOR KAWASAKI SYNDROME IN THE UNITED-STATES AND CANADA [J].

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