MYASTHENIA-GRAVIS AND MYASTHENIC SYNDROMES

[21] COMPLETE MESSENGER-RNA CODING SEQUENCE OF THE ACETYLCHOLINE BINDING ALPHA-SUBUNIT OF TORPEDO-MARMORATA ACETYLCHOLINE-RECEPTOR - A MODEL FOR THE TRANSMEMBRANE ORGANIZATION OF THE POLYPEPTIDE-CHAIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1983, 80 (07): : 2067 - 2071

[22] FUNCTIONAL ACTIVITIES OF AUTOANTIBODIES TO ACETYLCHOLINE-RECEPTORS AND THE CLINICAL SEVERITY OF MYASTHENIA-GRAVIS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 307 (13) : 769 - 775

[23] ACTIVATION AND DESENSITIZATION OF TORPEDO ACETYLCHOLINE-RECEPTOR - EVIDENCE FOR SEPARATE BINDING-SITES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1982, 79 (22): : 6757 - 6761

[24] NATURALLY-OCCURRING ANTI-IDIOTYPIC ANTIBODIES IN MYASTHENIA-GRAVIS PATIENTS [J]. NATURE, 1983, 301 (5901) : 611 - 614

[25] AN ENZYME-LINKED IMMUNOABSORBENT ASSAY FOR MEASURING ANTIBODIES AGAINST MUSCLE ACETYLCHOLINE-RECEPTOR [J]. JOURNAL OF IMMUNOLOGICAL METHODS, 1983, 57 (1-3) : 111 - 119

[26] Effect of eserine on neuromuscular transmission [J]. JOURNAL OF NEUROPHYSIOLOGY, 1942, 5 (03) : 211 - 230

[27] ELIAS SB, 1983, NEW ENGL J MED, V308, P402

[28] ENGEL AG, 1977, MAYO CLIN PROC, V52, P267

[29] A NEWLY RECOGNIZED CONGENITAL MYASTHENIC SYNDROME ATTRIBUTED TO A PROLONGED OPEN TIME OF THE ACETYLCHOLINE-INDUCED ION CHANNEL [J]. ANNALS OF NEUROLOGY, 1982, 11 (06) : 553 - 569

[30] NEW MYASTHENIC SYNDROME WITH ENDPLATE ACETYLCHOLINESTERASE DEFICIENCY, SMALL NERVE-TERMINALS, AND REDUCED ACETYLCHOLINE-RELEASE [J]. ANNALS OF NEUROLOGY, 1977, 1 (04) : 315 - 330

[21] COMPLETE MESSENGER-RNA CODING SEQUENCE OF THE ACETYLCHOLINE BINDING ALPHA-SUBUNIT OF TORPEDO-MARMORATA ACETYLCHOLINE-RECEPTOR - A MODEL FOR THE TRANSMEMBRANE ORGANIZATION OF THE POLYPEPTIDE-CHAIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1983, 80 (07): : 2067 - 2071

[22] FUNCTIONAL ACTIVITIES OF AUTOANTIBODIES TO ACETYLCHOLINE-RECEPTORS AND THE CLINICAL SEVERITY OF MYASTHENIA-GRAVIS [J]. NEW ENGLAND JOURNAL OF MEDICINE, 1982, 307 (13) : 769 - 775

[23] ACTIVATION AND DESENSITIZATION OF TORPEDO ACETYLCHOLINE-RECEPTOR - EVIDENCE FOR SEPARATE BINDING-SITES [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES, 1982, 79 (22): : 6757 - 6761

[24] NATURALLY-OCCURRING ANTI-IDIOTYPIC ANTIBODIES IN MYASTHENIA-GRAVIS PATIENTS [J]. NATURE, 1983, 301 (5901) : 611 - 614

[25] AN ENZYME-LINKED IMMUNOABSORBENT ASSAY FOR MEASURING ANTIBODIES AGAINST MUSCLE ACETYLCHOLINE-RECEPTOR [J]. JOURNAL OF IMMUNOLOGICAL METHODS, 1983, 57 (1-3) : 111 - 119

[26] Effect of eserine on neuromuscular transmission [J]. JOURNAL OF NEUROPHYSIOLOGY, 1942, 5 (03) : 211 - 230

[27] ELIAS SB, 1983, NEW ENGL J MED, V308, P402

[28] ENGEL AG, 1977, MAYO CLIN PROC, V52, P267

[29] A NEWLY RECOGNIZED CONGENITAL MYASTHENIC SYNDROME ATTRIBUTED TO A PROLONGED OPEN TIME OF THE ACETYLCHOLINE-INDUCED ION CHANNEL [J]. ANNALS OF NEUROLOGY, 1982, 11 (06) : 553 - 569

[30] NEW MYASTHENIC SYNDROME WITH ENDPLATE ACETYLCHOLINESTERASE DEFICIENCY, SMALL NERVE-TERMINALS, AND REDUCED ACETYLCHOLINE-RELEASE [J]. ANNALS OF NEUROLOGY, 1977, 1 (04) : 315 - 330