THE CURRENT MANAGEMENT OF VASCULAR BIRTHMARKS

被引:192
作者
ENJOLRAS, O
MULLIKEN, JB
机构
[1] Interdisciplinary Study Group for Vascular Malformations, Department of Neuroradiology, Hôpital Lariboisière
[2] Craniofacial Center, Division of Plastic Surgery, Children's Hospital
[3] Department of Dermatology, Hôpital Tarnier, Paris
[4] Harvard Medical School, Boston, Massachusetts
关键词
D O I
10.1111/j.1525-1470.1993.tb00393.x
中图分类号
R75 [皮肤病学与性病学];
学科分类号
100206 ;
摘要
Two vascular birthmarks are hemangiomas and vascular malformations. Hemangiomas grow by cellular proliferation. Their hallmark is rapid neonatal growth. Spontaneous regression begins when the infant is 6 to 1 0 months old, but it may continue until 8 to 1 0 years of age. Hemangiomas are infrequently life-threatening. Pharmacologic treatment is indispensible; unsightly sequelae require surgical treatment. Vascular malformations consist of dysplastic vessels and are present on a lifelong basis. They are either slow-flow (capillary, venous, lymphatic) or fast-flow anomalies with arteriovenous shunting. Complex combined vascular malformations are observed as well. Ten years ago angiographic studies clearly demonstrated the differences among the various lesions. Today a noninvasive diagnostic approach is recommended, particularly in children. Ultrasonography, Doppler flow imaging, and magnetic resonance imaging are the most informative techniques, revealing the extent of tissue involvement and differentiating fast-flow from slow-flow anomalies. Risks and management differ depending on the type of vascular malformation.
引用
收藏
页码:311 / 333
页数:23
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共 63 条
[41]  
Stevenson RF, Thomson HG, Morin JD, Unrecognized ocular problems associated with portwine stains of the face in children, Can Med Assoc J, 111, pp. 953-955, (1974)
[42]  
Diebler C, Dulac O, Syndrome de Sturge‐Weber, Pediatric neurology and neuroradiology, pp. 99-104, (1987)
[43]  
Jacoby CG, Yuh WT, Accelerated myelination in early Sturge‐Weber syndrome, J Comput Assist Tomogr, 11, pp. 226-231, (1987)
[44]  
Chiron C, Raynaud C, Tzourio N, Et al., Regional cerebral blood flow by SPECT imaging in SlurgeWeber disease, J Neurol Neurosurg Psychiatry, 52, pp. 1402-1409, (1989)
[45]  
Chugani HT, Maziotta JC, Phelps MI, Sturge‐Weber syndrome, a study of cerebral glucose utilization with positron emission tomography, J Pediatr, 114, pp. 244-253, (1989)
[46]  
Klap P, Hadjean E, Negrier B, Et al., Les angiomes capillaroveineux temporomasseterins, Ann Otolaryngol Chir Cervicofac, 104, pp. 433-439, (1987)
[47]  
Meyer JS, Hoffer FA, Barnes PD, Et al., MRI–‐correlation of the biological classification of soft tissue vascular anomalies, AJR, 157, pp. 559-564, (1991)
[48]  
Boukobza M, Guichard JP, Gelbert F, Et al., pp. 8-11, (1992)
[49]  
Riche MC, Hadjean E, Tran Ba Huy P, Et al., The treatment of capillary‐venous malformations using a new fibrosing agent, Plastic and Reconstructive Surgery, 71, pp. 607-612, (1983)
[50]  
Dubois JM, Sebag GH, De Prose Y, Et al., The treatment of soft tissue venous malformations in children. Percutaneous sclerotherapy with Ethibloc, Radiology, 180, pp. 195-198, (1991)