SIGMA(S)-DEPENDENT OVEREXPRESSION OF FTSZ IN AN ESCHERICHIA-COLI K-12 RPOB MUTANT THAT IS RESISTANT TO THE DIVISION INHIBITORS DICB AND DICF RNA

被引:7
作者
CAM, K
CUZANGE, A
BOUCHE, JP
机构
[1] Laboratoire de Microbiologie et Génétique moléculaire Centre National, de la Recherche Scientifique, Toulouse, 31062
来源
MOLECULAR & GENERAL GENETICS | 1995年 / 248卷 / 02期
关键词
ESCHERICHIA COLI K-12; RPOB; FTSZ; RPOS; CELL DIVISION;
D O I
10.1007/BF02190800
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
The Escherichia coli genes dicF and dicB encode division inhibitors, which prevent the synthesis and activity, respectively, of the essential division protein FtsZ. A mutation at the C-terminal end of the RNA polymerase beta subunit renders cells resistant to both inhibitors. In the mutant strain the level of the ftsZ gene product is higher than in the wild type. Disruption of rpoS, which encodes the stationary phase sigma factor sigma(S), lowers FtsZ protein levels in the mutant, and partially restores sensitivity to the inhibitors.
引用
收藏
页码:190 / 194
页数:5
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