FAMILIAL THYROID PEROXIDASE DEFECT

被引：11

作者：

MEDEIROSNETO, GA ^{[1
]}

OKAMURA, K ^{[1
]}

CAVALIERE, H ^{[1
]}

TAUROG, A ^{[1
]}

KNOBEL, M ^{[1
]}

BISI, H ^{[1
]}

KALLAS, WG ^{[1
]}

MATTAR, E ^{[1
]}

机构：

[1] UNIV TEXAS,HLTH SCI CTR,DEPT PHARMACOL,DALLAS,TX 75235

来源：

CLINICAL ENDOCRINOLOGY | 1982年 / 17卷 / 01期

关键词：

D O I：

10.1111/j.1365-2265.1982.tb02628.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1 / 14

页数：14

共 20 条

[11] DIFFERENTIATION OF 2 ABNORMALITIES IN THYROID PEROXIDASE CAUSING ORGANIFICATION DEFECT AND GOITROUS HYPOTHYROIDISM [J].

NIEPOMNISZCZE, H ;

ROSENBLOOM, AL ;

DEGROOT, LJ ;

SHIMAOKA, K ;

REFETOFF, S ;

YAMAMOTO, K .

METABOLISM-CLINICAL AND EXPERIMENTAL, 1975, 24 (01) :57-67

[12] ABNORMAL THYROID PEROXIDASE CAUSING IODIDE ORGANIFICATION DEFECT [J].

NIEPOMNISZCZE, H ;

HAGEN, GA ;

DEGROOT, LJ .

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1972, 34 (04) :607-+

[13] PEROXIDASE DEFECT IN CONGENITAL GOITER WITH COMPLETE ORGANIFICATION BLOCK [J].

NIEPOMNISZCZE, H ;

CASTELLS, S ;

DEGROOT, LJ ;

REFETOFF, S ;

KIM, OS ;

RAPOPORT, B ;

HATI, R .

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1973, 36 (02) :347-357

[14] CONGENITAL GOITER DUE TO THYROID PEROXIDASE-IODINASE DEFECT [J].

NIEPOMNISZCZE, H ;

COLEONI, AH ;

TARGOVNIK, HM ;

IORCANSKY, S ;

DEGROSSI, OJ .

ACTA ENDOCRINOLOGICA, 1980, 93 (01) :25-31

[15] DEFECTIVE THYROID PEROXIDASE SOLUBILIZED FROM A FAMILIAL GOITER WITH IODINE ORGANIFICATION DEFECT [J].

POMMIER, J ;

TOURNIAIRE, J ;

DEME, D ;

CHALENDAR, D ;

BORNET, H ;

NUNEZ, J .

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1974, 39 (01) :69-80

[16] VARIATIONS IN RESPONSE OF THYROID-GLAND OF RAT TO DIFFERENT LOW-IODINE DIETS - CORRELATION WITH IODINE CONTENT OF DIET [J].

RIESCO, G ;

TAUROG, A ;

LARSEN, PR .

ENDOCRINOLOGY, 1976, 99 (01) :270-280

[17]

SALVATOR.G, 1965, J BIOL CHEM, V240, P2935

[18] A STUDY OF A FAMILY OF GOITROUS CRETINS [J].

STANBURY, JB ;

HEDGE, AN .

JOURNAL OF CLINICAL ENDOCRINOLOGY, 1950, 10 (11) :1471-1484

[19]

STANBURY JB, 1972, METABOLIC BASIS INHE, P223

[20] LACK OF THYROID PEROXIDASE-ACTIVITY AS CAUSE OF CONGENITAL GOITROUS HYPOTHYROIDISM [J].

VALENTA, LJ ;

BODE, H ;

VICKERY, AL ;

CAULFIELD, JB ;

MALOOF, F .

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1973, 36 (05) :830-844

← 1 2 →