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H2 and H3 relaxin inhibit high glucose-induced apoptosis in neonatal rat ventricular myocytes[J] . Xiaohui Zhang,Xiao Ma,Meng Zhao,Bo Zhang,Jinyu Chi,Wenxiu Liu,Wenjia Chen,Yu Fu,Yue Liu,Xinhua Yin.Biochimie . 2015
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Diabetic cardiomyopathy: Pathophysiological mechanisms and cardiac dysfuntion[J] . Goyal,BR,Mehta,AA.Human and Experimental Toxicology . 2013 (6)
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The hyperglycemia stimulated myocardial endoplasmic reticulum (ER) stress contributes to diabetic cardiomyopathy in the transgenic non-obese type 2 diabetic rats: A differential role of unfolded protein response (UPR) signaling proteins[J] . Arun Prasath Lakshmanan,Meilei Harima,Kenji Suzuki,Vivian Soetikno,Masaki Nagata,Takashi Nakamura,Toshihiro Takahashi,Hirohito Sone,Hiroshi Kawachi,Kenichi Watanabe.International Journal of Biochemistry and Cell Bi . 2013 (2)
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Inflammation, Endoplasmic Reticulum Stress, Autophagy, and the Monocyte Chemoattractant Protein-1/CCR2 Pathway[J] . Pappachan E. Kolattukudy,Jianli Niu.Circulation Research . 2012 (1)
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Interrelationship Between Cardiac Hypertrophy, Heart Failure, and Chronic Kidney Disease: Endoplasmic Reticulum Stress As a Mediator of Pathogenesis[J] . Jeffrey G. Dickhout,Rachel E. Carlisle,Richard C. Austin.Circulation Research . 2011 (5)