Ciliary neurotrophic factor protects striatal output neurons in an animal model of Huntington disease

被引:100
作者
Anderson, KD
Panayotatos, N
Corcoran, TL
Lindsay, RM
Wiegand, SJ
机构
[1] Regeneron Pharmaceutical, Inc., Tarrytown
[2] Regeneron Pharmaceuticals, Inc., Tarrytown, NY 10591-6707
关键词
brain-derived neurotrophic factor; excitotoxicity; nerve growth factor; neurotrophin-3; quinolinic acid;
D O I
10.1073/pnas.93.14.7346
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
07 ; 0710 ; 09 ;
摘要
Huntington disease is a dominantly inherited, untreatable neurological disorder featuring a progressive loss of striatal output neurons that results in dyskinesia, cognitive decline, and, ultimately, death. Neurotrophic factors have recently been shown to be protective in several animal models of neurodegenerative disease, raising the possibility that such substances might also sustain the survival of compromised striatal output neurons. We determined whether intracerebral administration of brain-derived neurotrophic factor, nerve growth factor, neurotrophin-3, or ciliary neurotrophic factor could protect striatal output neurons in a rodent model of Huntington disease. Whereas treatment with brain-derived neurotrophic factor, nerve growth factor, or neurotrophin-3 provided no protection of striatal output neurons from death induced by intrastriatal injection of quinolinic acid, an N-methyl-D-aspartate glutamate receptor agonist, treatment with ciliary neurotrophic factor afforded marked protection against this neurodegenerative insult.
引用
收藏
页码:7346 / 7351
页数:6
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