共 28 条
Induction of differentiation of pre-NKT cells to mature Vα14 NKT cells by granulocyte macrophage colony-stimulating factor
被引:47
作者:

Sato, H
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机构: Chiba Univ, CREST, Chiba 2608670, Japan

Nakayama, T
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机构: Chiba Univ, CREST, Chiba 2608670, Japan

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Saito, Y
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机构: Chiba Univ, CREST, Chiba 2608670, Japan

Taniguchi, M
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机构: Chiba Univ, CREST, Chiba 2608670, Japan
机构:
[1] Chiba Univ, CREST, Chiba 2608670, Japan
[2] Chiba Univ, Dept Mol Immunol, Grad Sch Med, Chiba 2608670, Japan
[3] Chiba Univ, Sch Med, Dept Dev Immunol, Chiba 2608670, Japan
[4] Chiba Univ, Sch Med, Dept Internal Med 2, Chiba 2608670, Japan
来源:
关键词:
D O I:
10.1073/pnas.96.13.7439
中图分类号:
O [数理科学和化学];
P [天文学、地球科学];
Q [生物科学];
N [自然科学总论];
学科分类号:
07 ;
0710 ;
09 ;
摘要:
V alpha 14 NKT cells express an invariant antigen receptor encoded by Va14 and J alpha 281 gene segments as well as natural killer (NK) markers, including NK1.1. Here, we describe a precursor population of NKT cells (pre-NKT) that expresses NK1.1, T cell antigen receptor beta, pT alpha, and RAG1/2 but not V alpha 14 and surface CD3 epsilon. Such pre-NKT cells were differentiated successfully in vitro into mature CD3 epsilon(+) V alpha 14(+) NKT tells by IL-15 and granulocyte/macrophage colony-stimulating factor (GR;I-CSF) in conjunction with stroma cells. Interestingly, only GM-CSF without stroma cells induced the V alpha 14-J alpha 281 gene rearrangement in the pre-NKT cells. This also was confirmed by the findings that the number of mature V alpha 14 NKT cells and the frequency of V alpha 14-J alpha 281 rearrangements were decreased significantly in the mice lacking a GM-CSF receptor component, common beta-chain. These results suggest a crucial role of GM-CSF in the development of V alpha 14 NKT cells in vivo.
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页码:7439 / 7444
页数:6
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