Stress response, cell death and signalling: the many faces of reactive oxygen species

被引：131

作者：

Mahalingam, R

Fedoroff, N ^{[1
]}

机构：

[1] Penn State Univ, Dept Biol, University Pk, PA 16802 USA

[2] Penn State Univ, Life Sci Consortium, University Pk, PA 16802 USA

来源：

PHYSIOLOGIA PLANTARUM | 2003年 / 119卷 / 01期

关键词：

D O I：

10.1034/j.1399-3054.2003.00156.x

中图分类号：

Q94 [植物学];

学科分类号：

071001 ;

摘要：

Plants respond to pathogens and abiotic stresses by transient increases in the production of reactive oxygen species (ROS) and ion fluxes, which activate both local programmed cell death and systemic increases in stress- and pathogen-resistance. The present essay explores the emerging complexity of the multiple roles that ROS play in intra- and intercellular communication in both stressed and unstressed organisms.

引用

页码：56 / 68

页数：13

共 172 条

[51] Hydrogen peroxide from the oxidative burst is neither necessary nor sufficient for hypersensitive cell death induction, phenylalanine ammonia lyase stimulation, salicylic acid accumulation, or scopoletin consumption in cultured tobacco cells treated with elicitin [J].