A comprehensive guide to antibody and T-cell responses in type 1 diabetes

被引:134
作者
Lieberman, SM
DiLorenzo, TP
机构
[1] Yeshiva Univ Albert Einstein Coll Med, Dept Microbiol & Immunol, Bronx, NY 10461 USA
[2] Albert Einstein Coll Med, Dept Med, Div Endocrinol, Bronx, NY 10461 USA
来源
TISSUE ANTIGENS | 2003年 / 62卷 / 05期
关键词
antigens; autoimmunity; CD4 T cells; CD8 T cells; epitopes; NOD mice; type; 1; diabetes; beta cells;
D O I
10.1034/j.1399-0039.2003.00152.x
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
Type 1 diabetes (T1D) is an organ-specific autoimmune disease in which the insulin-producing beta cells in the pancreatic islets are selectively eliminated. T cells specific for beta-cell antigens are the mediators of this precise cellular destruction. However, antibodies to beta-cell proteins are also generated and may be used for predicting disease in at-risk populations. Over the past two decades, numerous beta-cell proteins and lipids have been implicated as autoantigens in patients or in non-obese diabetic (NOD) mice, a well-studied animal model of T1D. Here, we present a review of these antigens, accompanied by their T-cell epitopes, where known, and a discussion of our current understanding of why particular self-proteins become disease-inciting antigens. Although two dozen beta-cell antigens have been identified to date, few of these have been confirmed to be recognized by pathogenic T cells early in the disease process. Further identification and characterization of initiating beta-cell antigens targeted by pathogenic T cells should be a priority for future studies.
引用
收藏
页码:359 / 377
页数:19
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