Toward antiapoptosis as a new treatment modality

[1] TARGETED MUTATION IN THE FAS GENE CAUSES HYPERPLASIA IN PERIPHERAL LYMPHOID ORGANS AND LIVER [J]. NATURE GENETICS, 1995, 11 (03) : 294 - 300

[2] The Bcl-2 protein family: Arbiters of cell survival [J]. SCIENCE, 1998, 281 (5381) : 1322 - 1326

[3] Enhanced Gαq signaling:: A common pathway mediates cardiac hypertrophy and apoptotic heart failure [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (17) : 10140 - 10145

[4] Oxidative stress activates extracellular signal-regulated kinases through Src and ras in cultured cardiac myocytes of neonatal rats [J]. JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (07) : 1813 - 1821

[5] Ventricular myocytes are not terminally differentiated in the adult mammalian heart [J]. CIRCULATION RESEARCH, 1998, 83 (01) : 1 - 14

[6] The delivery of angiogenic factors to the heart by microsphere therapy [J]. NATURE BIOTECHNOLOGY, 1998, 16 (02) : 159 - 162

[7] Bardales RH, 1996, AM J PATHOL, V149, P821

[8] CARDIOPROTECTIVE EFFECT OF INSULIN-LIKE GROWTH-FACTOR-I IN MYOCARDIAL-ISCHEMIA FOLLOWED BY REPERFUSION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (17) : 8031 - 8035

[9] Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development [J]. CELL, 1998, 94 (06) : 727 - 737

[10] STRETCH-INDUCED PROGRAMMED MYOCYTE CELL-DEATH [J]. JOURNAL OF CLINICAL INVESTIGATION, 1995, 96 (05) : 2247 - 2259

[1] TARGETED MUTATION IN THE FAS GENE CAUSES HYPERPLASIA IN PERIPHERAL LYMPHOID ORGANS AND LIVER [J]. NATURE GENETICS, 1995, 11 (03) : 294 - 300

[2] The Bcl-2 protein family: Arbiters of cell survival [J]. SCIENCE, 1998, 281 (5381) : 1322 - 1326

[3] Enhanced Gαq signaling:: A common pathway mediates cardiac hypertrophy and apoptotic heart failure [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (17) : 10140 - 10145

[4] Oxidative stress activates extracellular signal-regulated kinases through Src and ras in cultured cardiac myocytes of neonatal rats [J]. JOURNAL OF CLINICAL INVESTIGATION, 1997, 100 (07) : 1813 - 1821

[5] Ventricular myocytes are not terminally differentiated in the adult mammalian heart [J]. CIRCULATION RESEARCH, 1998, 83 (01) : 1 - 14

[6] The delivery of angiogenic factors to the heart by microsphere therapy [J]. NATURE BIOTECHNOLOGY, 1998, 16 (02) : 159 - 162

[7] Bardales RH, 1996, AM J PATHOL, V149, P821

[8] CARDIOPROTECTIVE EFFECT OF INSULIN-LIKE GROWTH-FACTOR-I IN MYOCARDIAL-ISCHEMIA FOLLOWED BY REPERFUSION [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (17) : 8031 - 8035

[9] Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development [J]. CELL, 1998, 94 (06) : 727 - 737

[10] STRETCH-INDUCED PROGRAMMED MYOCYTE CELL-DEATH [J]. JOURNAL OF CLINICAL INVESTIGATION, 1995, 96 (05) : 2247 - 2259