MOLECULAR ORIGINS OF CANCER DNA Damage, Aging, and Cancer

[1] Cytotoxicity and mutagenicity of endogenous DNA base lesions as potential cause of human aging [J]. MECHANISMS OF AGEING AND DEVELOPMENT, 2008, 129 (7-8) : 353 - 365

[2] Eukaryotic DNA damage tolerance and translesion syntheses through covalent modifications of PCNA [J]. CELL RESEARCH, 2008, 18 (01) : 162 - 173

[3] Transcription-coupled repair and premature ageing [J]. MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS, 2005, 577 (1-2) : 179 - 194

[4] [Anonymous], 2005, DNA REPAIR MUTAGENES

[5] A synthetic lethal therapeutic approach: Poly(ADP) ribose polymerase inhibitors for the treatment of cancers deficient in DNA double-strand break repair [J]. JOURNAL OF CLINICAL ONCOLOGY, 2008, 26 (22) : 3785 - 3790

[6] Repair and genetic consequences of endogenous DNA base damage in mammalian cells [J]. ANNUAL REVIEW OF GENETICS, 2004, 38 : 445 - 476

[7] DNA damage signalling guards against activated oncogenes and tumour progression [J]. ONCOGENE, 2007, 26 (56) : 7773 - 7779

[8] Impact of reduced insulin-like growth factor-1/insulin signaling on aging in mammals: novel findings [J]. AGING CELL, 2008, 7 (03) : 285 - 290

[9] Poly(ADP-ribosyl)ation in mammalian ageing [J]. NUCLEIC ACIDS RESEARCH, 2007, 35 (22) : 7456 - 7465

[10] Human cancers express a mutator phenotype (vol 103, pg 18238, 2006) [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2006, 103 (51) : 19605 - 19605

[1] Cytotoxicity and mutagenicity of endogenous DNA base lesions as potential cause of human aging [J]. MECHANISMS OF AGEING AND DEVELOPMENT, 2008, 129 (7-8) : 353 - 365

[2] Eukaryotic DNA damage tolerance and translesion syntheses through covalent modifications of PCNA [J]. CELL RESEARCH, 2008, 18 (01) : 162 - 173

[3] Transcription-coupled repair and premature ageing [J]. MUTATION RESEARCH-FUNDAMENTAL AND MOLECULAR MECHANISMS OF MUTAGENESIS, 2005, 577 (1-2) : 179 - 194

[4] [Anonymous], 2005, DNA REPAIR MUTAGENES

[5] A synthetic lethal therapeutic approach: Poly(ADP) ribose polymerase inhibitors for the treatment of cancers deficient in DNA double-strand break repair [J]. JOURNAL OF CLINICAL ONCOLOGY, 2008, 26 (22) : 3785 - 3790

[6] Repair and genetic consequences of endogenous DNA base damage in mammalian cells [J]. ANNUAL REVIEW OF GENETICS, 2004, 38 : 445 - 476

[7] DNA damage signalling guards against activated oncogenes and tumour progression [J]. ONCOGENE, 2007, 26 (56) : 7773 - 7779

[8] Impact of reduced insulin-like growth factor-1/insulin signaling on aging in mammals: novel findings [J]. AGING CELL, 2008, 7 (03) : 285 - 290

[9] Poly(ADP-ribosyl)ation in mammalian ageing [J]. NUCLEIC ACIDS RESEARCH, 2007, 35 (22) : 7456 - 7465

[10] Human cancers express a mutator phenotype (vol 103, pg 18238, 2006) [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2006, 103 (51) : 19605 - 19605