Manipulation of the host cell death pathway by Shigella

被引：26

作者：

Ashida, Hiroshi ^{[1
]}

Kim, Minsoo ^{[1
]}

Sasakawa, Chihiro ^{[1
,2
,3
]}

机构：

[1] Univ Tokyo, Inst Med Sci, Div Bacterial Infect Biol, Tokyo 1088639, Japan

[2] Nippon Inst Biol Sci, Tokyo 1980024, Japan

[3] Chiba Univ, Med Mycol Res Ctr, Chiba 2608673, Japan

来源：

CELLULAR MICROBIOLOGY | 2014年 / 16卷 / 12期

关键词：

NONCANONICAL INFLAMMASOME ACTIVATION; III SECRETION; PROGRAMMED NECROSIS; BACTERIAL LIGANDS; AIM2; INFLAMMASOME; APOPTOSIS; CASPASE-1; FLEXNERI; INNATE; EFFECTOR;

D O I：

10.1111/cmi.12367

中图分类号：

Q2 [细胞生物学];

学科分类号：

071013 [干细胞生物学];

摘要：

Host cells deploy multiple defences against microbial infection. One prominent host defence mechanism, the death of infected cells, plays a pivotal role in clearing damaged cells, eliminating pathogens, removing replicative niches, exposing intracellular bacterial pathogens to extracellular immune surveillance and presenting bacteria-derived antigens to the adaptive immune system. Although cell death can occur under either physiological or pathophysiological conditions, it acts as an innate defence mechanism against bacterial pathogens by limiting their persistent colonization. However, many bacterial pathogens, including Shigella, have evolved mechanisms that manipulate host cell death for their own benefit.

引用

页码：1757 / 1766

页数：10

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