Interaction between human MCM7 and Rad17 proteins is required for replication checkpoint signaling
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作者:
Tsao, CC
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Burnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USABurnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USA
Tsao, CC
[1
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Geisen, C
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Burnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USABurnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USA
Geisen, C
[1
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Abraham, RT
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Burnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USABurnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USA
Abraham, RT
[1
]
机构:
[1] Burnham Inst, Proram Signal Transduct Res, Ctr Canc Res, La Jolla, CA 92037 USA
Human Rad17 (hRad17) is centrally involved in the activation of cell-cycle checkpoints by genotoxic agents or replication stress. Here we identify hMCM7, a core component of the DNA replication apparatus, as a novel hRad17-interacting protein. In HeLa cells, depletion of either hRad17 or hMCM7 with small-interfering RNA suppressed ultraviolet (UV) light- or aphidicolin-induced hChk1 phosphorylation, and abolished UV-induced S-phase checkpoint activation. Similar results were obtained after transfection of these cells with a fusion protein containing the hMCM7-binding region of hRad17. The hMCM7-depleted cells were also defective for the formation of ATR-containing nuclear foci after UV irradiation, suggesting that hMCM7 is required for stable recruitment of ATR to damaged DNA. These results demonstrate that hMCM7 plays a direct role in the transmission of DNA damage signals from active replication forks to the S-phase checkpoint machinery in human cells.
机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Bermudez, VP
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Lindsey-Boltz, LA
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Lindsey-Boltz, LA
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Cesare, AJ
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Cesare, AJ
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Maniwa, Y
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Maniwa, Y
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Griffith, JD
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Griffith, JD
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Hurwitz, J
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Hurwitz, J
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Sancar, A
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Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USAMem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Bermudez, VP
;
Lindsey-Boltz, LA
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Lindsey-Boltz, LA
;
Cesare, AJ
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Cesare, AJ
;
Maniwa, Y
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Maniwa, Y
;
Griffith, JD
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Griffith, JD
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Hurwitz, J
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机构:Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA
Hurwitz, J
;
Sancar, A
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Mem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USAMem Sloan Kettering Canc Ctr, Program Mol Biol, Sloan Kettering Inst, New York, NY 10021 USA